FKBP5 polymorphisms induce differential glucocorticoid responsiveness in primary CNS cells - First insights from novel humanized mice.

For Tsc22d3 , the ANOVA of ranks suggested a highly significant interaction of cell type and dose, F (8, 237) = 38.0, p < 0.0001, which was attributable post hoc to both glial cell types differing from neurons (2.73 ± 2.77).

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The cycle difference after stimulation negatively correlated with the basal expression of Fkbp5 for the genes Nfkbia (β = −0.46, p = 0.006), Tsc22d3 (β = −0.42, p = 0.01), and Sgk1 (β = −0.34, p = 0.05) and was marginally significant for Nr3c1 , Sesn1 , and Fkbp5 .

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