<i>Aurka</i> deficiency in the intestinal epithelium promotes age-induced obesity via propionate-mediated AKT activation.

Unexpectedly, there was a decreasing trend in glucose levels in Aurka f/+ ;Villin Cre/+ mice (Figure 2 M), suggesting that Aurka loss in the intestinal epithelium could influence metabolism, thus contributing to obesity rather than glucose tolerance.

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