Although several studies have shown that nAbs are the best correlate of protection against measles, 31 , 32 there may be significant in vivo contributions from antibody-dependent cellular cytotoxicity (ADCC), complement-dependent cytolysis, and phagocytosis of opsonized virions.
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Serotypic evolution of measles virus is constrained by multiple co-dominant B cell epitopes on its surface glycoproteins.
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