Alterations of the Platelet Proteome in Lung Cancer: Accelerated F13A1 and ER Processing as New Actors in Hypercoagulability.

Once again, the F13A1 level of spot 12c (pI 5.85) attracted attention in the opposite direction by a weak trend of a negative correlation with enzymatic F13A1 activity ( Table 3 ).

also in 1,404 other papers

Remarkably, in patients with lung cancer as well as in LA-positive patients with thrombosis history [ 16 ] we identified a highly significant increase in the PDI family member, P4HB, in platelets, which also manifest this ER chaperone as a highly purposive antithrombotic drug target.

also in 132,142 other papers