Lack of WWC2 Protein Leads to Aberrant Angiogenesis in Postnatal Mice.

Evaluation of vascularised and hypersprouting areas revealed that a knock-out of the WWC2 gene leads to a slight decrease of vascularised area compared to total retinal area and to a clear and highly significant increase of hypersprouting area compared to both total retinal area and vascularised area ( Figure 3 ).

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