In particular, there was a highly significant enrichment of genes affected by mTOR inhibitor rapamycin, highlighting the prominent role of lncTNK2-2:1 and GMDS-AS1 in the post-transcriptional regulation due to bimiralisib exposure.
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Exon-Intron Differential Analysis Reveals the Role of Competing Endogenous RNAs in Post-Transcriptional Regulation of Translation.
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