A myostatin-CCL20-CCR6 axis regulates Th17 cell recruitment to inflamed joints in experimental arthritis.

Sci Rep · 2021 · PMC8266846 · PMID 34239010

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Interestingly, analysis of corresponding CCL20 secretion revealed a highly significant reduction in CCL20 levels after inhibition of SMAD2 activation (Fig. 2 h), indicating that the myostatin-mediated increase in CCL20 secretion depends exclusively on activation of the SMAD pathway.

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