Prevalence of herpes virus in chronic periodontitis patients with and without type 2 diabetes mellitus: A clinico-microbiological study.

This altered immune response ultimately results in accumulation of high concentration of viral and bacterial substrate.[ 27 , 28 ] Subgingival samples were collected from the deepest pocket site, as the destruction and progression of periodontitis are active at the supra- and subgingival region.[ 29 ] Further, the site-specific and average PII, GI and PPD values of the healthy group (Group I) were highly significant to the chronic periodontitis patients (Groups II and III).

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