Methylation in response to injury and SAM in the frontal cortex Both neuropathic pain and SAM treatment are capable of inducing widespread and highly significant changes in frontal cortex methylation, with each condition inducing differential methylation in nearly 17% of the ∼22,500 genes examined.
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The methyl donor S-adenosyl methionine reverses the DNA methylation signature of chronic neuropathic pain in mouse frontal cortex.
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