7) Given the (perhaps not quite statistically significant) enhancement of myocarditis by the R296T mutant, it would be interesting to have some more data on the 41 genes solely induced by this mutant (Fig. 9) – do these give any indication as to a potential phenotype?
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The multi-functional reovirus σ3 protein is a virulence factor that suppresses stress granule formation and is associated with myocardial injury.
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