While, e.g., E or C inhibited the cell viability of NCI-H295R cells in a highly significant (E 180 uM: 2.5%; C 160 μM: 17%) and dose-dependent manner, even at extraordinarily high drug concentrations, cell viability remained high for MUC-1 (E 180 μM: 67%; C 160 μM: 70%; both p < 0.001 vs.
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Novel Insights into the Molecular Regulation of Ribonucleotide Reductase in Adrenocortical Carcinoma Treatment.
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