Interferon receptor-deficient mice are susceptible to eschar-associated rickettsiosis.

We also thank the reviewers for describing the study as “highly significant,” “rigorous and reliable as described and can be reproduced by others,” and as “relevant to investigators working in the field of rickettsial diseases and to a broader audience studying mechanisms of intracellular parasitism and host responses.” As proposed in our revision plan and suggested by all three reviewers, we have now performed histology in infected wild type and in interferon receptor-deficient mice at multiple time points of multiple organs.

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