Early Post-ischemic Brain Glucose Metabolism Is Dependent on Function of TLR2: a Study Using [¹⁸F]F-FDG PET-CT in a Mouse Model of Cardiac Arrest and Cardiopulmonary Resuscitation.

As previously mentioned, Zhang and colleagues discovered a rise in glucose uptake 72 h post-CA with significance in the hindbrain; in contrast, the increase in the regions of the forebrain failed to reach significance [ 33 ] corresponding to our results presented for the immediate period after ROSC.

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In comparison, baseline measurements of both mouse strains show a highly significant difference with regard to the absolute glucose SUV in the whole brain.

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