3 B demonstrate that while pretreatment with control EXu has no effect on the viral load, pretreating the cells with EXi-RVFV results in a highly significant decrease in intracellular RVFV levels.
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Exosomes originating from infection with the cytoplasmic single-stranded RNA virus Rift Valley fever virus (RVFV) protect recipient cells by inducing RIG-I mediated IFN-B response that leads to activation of autophagy.
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