S3D-I), and neutrophils (borderline significant, not shown), suggesting altered host immunity and dysregulated TME architecture, as also indicated by the downregulation of genes and pathways involved in, e.g., focal adhesion, regulation of actin cytoskeleton, and ECM-receptor interaction, that were identified in our DGE and KEGG pathway analyses, respectively.
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Microbiota of the prostate tumor environment investigated by whole-transcriptome profiling.
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