Hippler et al., have also recently utilized different IL17A reporter mice to show that neutrophils also do not produce IL17A during infection with Candida albicans 43 , suggesting that although neutrophils can be producers of IFNγ 44 , they may not be significant IL17A producers. αβ T cells are recognized as important mediators of pathology in HP, and IL17A has been established as the predominant cytokine in SR-induced HP 27 , 28 , 30 .
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ITK independent development of Th17 responses during hypersensitivity pneumonitis driven lung inflammation.
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