While the increase in food intake in terms of calories is unlikely to produce a large effect on homeostatic regulation of feeding, it is highly significant from a pharmacological perspective and for drug development studies, considering that this effect occurs during the time that blood and brain nicotine concentrations are at peak levels.
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Microstructural meal pattern analysis reveals a paradoxical acute increase in food intake after nicotine despite its long-term anorexigenic effects.
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