However, inhibition of miR-200a in uninjured spinal cords led to a twofold increase in brachyury expression (2.236 TPM), while the combination of miR-200a inhibition during injury led to a highly significant sevenfold increase in brachyury mRNA levels (5.8 TPM, Table S1 ).
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Regulation of stem cell identity by miR-200a during spinal cord regeneration.
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