The voltage-gated K+ channel Kv1.3 modulates platelet motility and α2β1 integrin-dependent adhesion to collagen.

Platelets · 2022 · PMC8935947 · PMID 34348571

Europe PMC DOI

hedged sentence

0.0519

closest p · 1.0× alpha

0.0519

boldest claim

The sentences

failed to reach significanceP = .0519so close (0.05 < p ≤ 0.1)

Following perfusion over the collagen peptides VWF-III and GFOGER a trend toward fewer Kv1.3 −/- platelets reaching the ruffled stage was observed, although this failed to reach significance ( P = .0519; Figure 3A ), and there was no difference in the percentage of platelets at each stage of platelet adhesion following perfusion over VWF-III and CRP-XL ( Figure 3D ).

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Quoted from the open-access full text in Europe PMC under the licence the publisher applied. The sentence is reproduced exactly as published; the emphasis is ours.

The voltage-gated K&lt;sup&gt;+&lt;/sup&gt; channel Kv1.3 modulates platelet motility and α&lt;sub&gt;2&lt;/sub&gt;β&lt;sub&gt;1&lt;/sub&gt; integrin-dependent adhesion to collagen.

The sentences

The voltage-gated K<sup>+</sup> channel Kv1.3 modulates platelet motility and α<sub>2</sub>β<sub>1</sub> integrin-dependent adhesion to collagen.