Furthermore, the level of neuronal acetylcholine receptor subunit beta-2 (CHRNB2) showed an increasing trend from hypoxia to reoxygenation ( Figure 6 ), indicating a compensatory effect in response to the reduced synthesis of acetylcholine.
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Tandem Mass Tagging-Based Quantitative Proteomics Analysis Reveals Damage to the Liver and Brain of <i>Hypophthalmichthys molitrix</i> Exposed to Acute Hypoxia and Reoxygenation.
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In the present study, we observed that ACh levels showed a decreasing trend during hypoxia and reoxygenation, which was in accordance with previous reports in mammals [ 39 ].