UBIAD1 alleviates ferroptotic neuronal death by enhancing antioxidative capacity by cooperatively restoring impaired mitochondria and Golgi apparatus upon cerebral ischemic/reperfusion insult.

Cell Biosci · 2022 · PMC8981649 · PMID 35379328

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may be significantno p-value reported

Moreover, our data suggests that the mitochondria and Golgi apparatus, which are critical subcellular organelles, may be significant treatment targets for ischemic stroke-induced ferroptosis.

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