The increased disruption in BCR-ABL T315I sessile patterning was highly significant compared to BCR-ABL P210 and was consistent with the higher increase in circulating hemocytes (Figure 1E) .
← all excerpts
Expression of chronic myeloid leukemia oncogenes BCR-ABL <sup>P210</sup> and BCR-ABL <sup>T315I</sup> affect cellular and humoral innate immunity in <i>Drosophila melanogaster</i>.
1
—
—