Despite a small number of patients was included in this cohort, they were homogeneously distributed between short-term and long-term responders, and the association between SEMA6A expression and recurrence-free interval was highly significant and high SEMA6A expression was detected in all the short-term responders.
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SEMA6A/RhoA/YAP axis mediates tumor-stroma interactions and prevents response to dual BRAF/MEK inhibition in BRAF-mutant melanoma.
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