Importantly, previous genome-wide gene expression profiling experiments conclusively demonstrated highly significant associations of increased likelihood of cancer patients’ death after therapy with acquisition by cancer cells of the stemness state [ 32 , 33 , 34 , 35 ].
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Effects of Anticancer Agent P-bi-TAT on Gene Expression Link the Integrin Thyroid Hormone Receptor to Expression of Stemness and Energy Metabolism Genes in Cancer Cells.
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