Arterial elastance (Ea) was also increased with SuCH and presented a nonsignificant trend toward reduction with treprostinil (mmHg/μL: 0.2 ± 0.002, 0.5 ± 0.09, and 0.2 ± 0.09, for normoxia, SuCH, and SuCH+Trep., respectively, Figure 3 B).
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Reversal of Right Ventricular Hypertrophy and Dysfunction by Prostacyclin in a Rat Model of Severe Pulmonary Arterial Hypertension.
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