Hesperidin abrogates bisphenol A endocrine disruption through binding with fibroblast growth factor 21 (FGF-21), α-amylase and α-glucosidase: an in silico molecular study.

Interestingly, our investigation revealed that hesperidin is a non-substrate and non-inhibitor of P-glycoprotein, thus a strong pharmacological advantage to hesperidin as there may not be significant alterations in its therapeutic effects due to the activity of P-glycoprotein when they are administered into the body.

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