In summary, our study shows that a gyrB mutation, even located outside of the QRDR and conferring a barely significant resistance level, can significantly increase persistence against ciprofloxacin.
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Repeated Exposure of <i>Escherichia coli</i> to High Ciprofloxacin Concentrations Selects <i>gyrB</i> Mutants That Show Fluoroquinolone-Specific Hyperpersistence.
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