Reduced Immunity Regulator MAVS Contributes to Non-Hypertrophic Cardiac Dysfunction by Disturbing Energy Metabolism and Mitochondrial Homeostasis.

Consistently, mitochondrial function did not differ between groups, although there was an increasing trend of mitochondrial gene expression resulting from MAVS loss, which might be transient compensation ( Figure 7D ).

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However, LV mass was not significantly different between WT and MAVS -/- mice, with only a slight trend toward an increase.

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