In our experiments, all five tested β-subunits (β2a, β2d, β2e, β3, β4) caused a robust and highly significant (6–12-fold; Figure 1A , Table 1 ) increase in current densities.
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β2-subunit alternative splicing stabilizes Cav2.3 Ca<sup>2+</sup> channel activity during continuous midbrain dopamine neuron-like activity.
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