Targeting mAKAPβ expression as a therapeutic approach for ischemic cardiomyopathy.

While the improvement in atrial hypertrophy did not quite reach statistical significance ( Figure 4g ), wet lung weight, an assay for pulmonary edema and a marker for heart failure, was significantly less after MI for AAV9sc.shmAKAP-treated mice and was similar to that for the sham-operated cohorts ( Figure 4h ).

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