The human long noncoding RNAs CoroMarker, MALAT1, CDR1as, and LINC00460 in whole blood of individuals after controlled short-term exposure with ultrafine metal fume particles at workplace conditions, and in human macrophages in vitro.

These local inflammatory responses in the upper airways preceded and accompanied significant systemic inflammatory responses characterized by a highly significant early increase of the acute phase mediator IL-6 at 10 h and significant increases of CRP and SAA at 29 h after exposure start [ 9 – 12 ].

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