Cerebral infarction-induced brain injury is associated with inflammatory responses, which play a critical role in neuron dysfunction and apoptosis in the brain regions, and the anti-inflammatory therapy might be significant in cerebral infarction [ 20 ].
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HMGA2 Promotes Brain Injury in Rats with Cerebral Infarction by Activating TLR4/NF-<i>κ</i>B Signaling Pathway.
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