Using a marker for the mature myelinating state (MBP), we continued to observe a decrease in mature Q84 oligodendrocytes at DIV3 with an increasing trend in immature oligodendrocytes (SMOC1+; MBP− cells) ( Figure 1 F,G).
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Pathogenetic Mechanisms Underlying Spinocerebellar Ataxia Type 3 Are Altered in Primary Oligodendrocyte Culture.
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