To improve patients' outcomes, it is highly significant to delve into the nosogenesis of AML and its treatment. miRNAs, as a kind of highly conserved 20-24 nucleotide length-long noncoding RNAs, are capable of binding to the incomplete complementary locus in mRNA 3′UTR to inhibit gene expression after transcription [ 27 ].
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miR-342-3p Inhibits Acute Myeloid Leukemia Progression by Targeting SOX12.
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