Genome-wide genotype-serum proteome mapping provides insights into the cross-ancestry differences in cardiometabolic disease susceptibility.

4 ) by excluding the participants with missing data in each protein or peptide and found that the results were largely similar to the main model with imputation, whereof only 11 pQTLs failed to reach significance at FDR < 0.05 (8 cis -acting variants, 3 trans -acting variants) (Supplementary Data 3 ).

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