Melatonin Ameliorates Diquat-Induced Testicular Toxicity via Reducing Oxidative Stress, Inhibiting Apoptosis, and Maintaining the Integrity of Blood-Testis Barrier in Mice.

As expected, exposure to diquat resulted in a highly significant decrease in sperm count ( Figure 1 G) and abnormal sperm morphology (Figure I) compared with control mice, suggesting that the spermatogenic function of the testes was compromised.

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