The findings imply that enhanced renal ketogenesis due to Hmgcs2 overexpression may be significant in the pathogenesis of diabetic neuropathy DN in patients with type 2 diabetes, indicating that Hmgcs2 is a potential therapeutic target for the management of diabetic renal complications [ 19 ].
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Multi-Omics Analyses Reveal the Mechanisms of Early Stage Kidney Toxicity by Diquat.
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