coli F18ac infection, while a decreasing trend was showed after the addition of L. reuteri ( Supplementary Table S5 ).
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Integrative ATAC-seq and RNA-seq analyses of IPEC-J2 cells reveals porcine transcription and chromatin accessibility changes associated with <i>Escherichia coli</i> F18ac inhibited by <i>Lactobacillus reuteri</i>.
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