GABAergic synapses onto SST and PV interneurons in the CA1 hippocampal region show cell-specific and integrin-dependent plasticity.

In the case of SST + interneurons a trend toward mIPSC frequency reduction upon RRETAWA treatment was apparent, but this effect did not reach statistical significance (before: 1.26 ± 0.71 Hz, after: 0.73 ± 0.17 Hz, n = 5, p = 0.087, Fig. 3 e–f).

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We found that in the case of SST + INs NMDA treatment resulted in a clear mIPSC amplitude potentiation which developed in time reaching a highly significant difference with respect to baseline recordings (SST + INs: 122 ± 5%, n = 9, relative to baseline, before NMDA: 45.3 ± 3.8 pA, 16–18 min. after the end of NMDA application: 54.3 ± 4 pA, p = 0.003, Fig. 4 a–d).

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