However, in co-silencing experiments, the reduction of IL-2 upon NCK-1 silencing failed to reach statistical significance; this may be due to the residual NCK-1 protein that may still play its function ( Supplementary Materials 6C, D, F ).
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Wiskott-Aldrich syndrome protein interacts and inhibits diacylglycerol kinase alpha promoting IL-2 induction.
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