Genome-wide mRNA profiling in urinary extracellular vesicles reveals stress gene signature for diabetic kidney disease.

In support to the pathophysiological roles of the stress score genes, pathway enrichment (gene ontology) analysis of all nominally significant differentially expressed genes between T1D macro- and normoalbuminuria groups also suggested changes in oxidation-reduction as well as transporter and EV-linked activities (p < 0.05, logFC>1, n = 130 genes, Table S8 ).

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