In other words, TBK1 phosphorylation induced by the virus, representative of enzyme activity, showed a significant trend toward an increase under the influence of β-glucan ( Figure 3 A,B), suggesting that the trained immunity induced by β-glucan is likely to be related to the post-translational modification of TBK1.
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β-Glucan Induces Training Immunity to Promote Antiviral Activity by Activating TBK1.
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