Through pairwise differential abundance analyses of sample groups, an increasing trend of Rikenellaceae, Pseudomonas and Fusobacterium, and decreasing trends of Staphylococcus , Actinobacillus and Gemmiger were observed in CRC, while Staphylococcus and Bifidobacterium were decreased in patients with only adenomas.
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Mucosal Microbiota from Colorectal Cancer, Adenoma and Normal Epithelium Reveals the Imprint of <i>Fusobacterium nucleatum</i> in Cancerogenesis.
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The pathways listed below appear to have a lower (mildly significant) predicted abundance in the Tumor group as compared to the normal control one: ectoine biosynthesis (MetaCyc identifier: P101-PWY), octane oxidation (P221-PW, myo-inositol degradation I (P562-PWY), catechol degradation to 2-hydroxypentadienoate II (PWY-5419), catechol degradation II (meta-cleavage