Furthermore, liraglutide ameliorated the impaired glucose tolerance caused by DHEA in mice, and semaglutide showed a trend toward ameliorating glucose tolerance ( Figure 1G ).
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Glucagon-like peptide-1 receptor agonists decrease hyperinsulinemia and hyperandrogenemia in dehydroepiandrosterone-induced polycystic ovary syndrome mice and are associated with mitigating inflammation and inducing browning of white adipose tissue†.
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