Formyl Peptide Receptor Type 2 Deficiency in Myeloid Cells Amplifies Sepsis-Induced Cardiac Dysfunction.

hFPR2 Deficiency in Myeloid Cells Aggravates the Host Immune Response to Polymicrobial Sepsis in Peritoneum and Increases Bacterial Load Analysis of peritoneal exudates by flow cytometry showed a trend for increased peritoneal Ly6G + CD64 − neutrophils at 24 h post-CLP in myeloid cell-specific hFPR2 KO CLP mice compared to hFPR2 CLP mice ( p = 0.09; Fig. 3 a, b), while CD64 + Ly6G − monocytes/macrophages were significantly decreased ( Fig. 3 a, c).

also in 53,322 other papers