Iron deficiency is related to lower muscle mass in community-dwelling individuals and impairs myoblast proliferation.

During a mitochondrial stress test, DFO treatment resulted in a small and borderline significant decline (−9%, P = 0.07) in ATP‐synthase linked respiration reflected by the difference between basal respiration rate and remaining respiration rate after oligomycin injection (OCR basal − OCR oligomycin ), which was not restored by co‐treatment with 10 μM FC (Figure 4B ).

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ECAR during basal respiration, ATP‐linked respiration and maximal respiration, reflecting lactate metabolism, increased after addition of DFO and decreased after additional treatment with FC, although these differences did not reach statistical significance (Figure 4D ).

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