Prefrontal influences on the function of the neural circuitry underlying anxious temperament in primates.

A repeated-measures ANOVA (group × time), controlling for age, demonstrated a group × time interaction that trended toward but did not reach statistical significance ( F (1,17) = 2.93, P = 0.11, partial \documentclass[12pt]{minimal} \usepackage{amsmath} \usepackage{wasysym} \usepackage{amsfonts} \usepackage{amssymb} \usepackage{amsbsy} \usepackage{upgreek} \usepackage{mathrsfs} \setlength{\oddsidemargin}{-69pt} \begin{document} $\upeta$\end{document} 2 = 0.15, Fig. 1e ), such that the lesioned animals demonstrated a more substantial decrease in AT relative to controls.

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To further characterize volumetric changes linked to the lesion, we assessed whole brain GMP and there were highly significant reductions in tissue volume in the targeted posterior OFC region.

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While relatively modest in magnitude and in general, trending toward significance, it is noteworthy that reductions were observed across several different threat-related paradigms.

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