Endothelial-derived FABP4 constitutes the majority of basal circulating hormone and regulates lipolysis-driven insulin secretion.

In BAT, Endo-KO mice showed a trend toward decreased FABP4 protein, and Myeloid-KO showed significantly reduced FABP4 compared with WT ( Supplemental Figure 3B and Supplemental Figure 4B ), suggesting some contribution of myeloid cells to BAT FABP4 at the whole-tissue level.

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