Fibulin7 Mediated Pathological Cardiac Remodeling through EGFR Binding and EGFR-Dependent FAK/AKT Signaling Activation.

Although FBLN7 overexpression also led to a more critical deposition of collagen in the infarct area, the difference in collagen I and III levels in the infarcted zone between FBLN7‐overexpressing mice and AAV‐NC‐injected mice did not reach statistical significance.

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