Differential Expression of the β3 Subunit of Voltage-Gated Ca²⁺ Channel in Mesial Temporal Lobe Epilepsy.

Our IHC finding is also consistent with the near-significant result from Western blot analysis (Fig. 4 A), and since both analyses represent different non-epilepsy control subject tissue samples, it gave us an enhanced confidence that the increased CACNB3 expression levels in mTLE temporal lobe neocortex were a true biological finding.

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